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Barack Obama
Calls For Universal Health Care Coverage
By NEDRA PICKLER
Associated Press
WASHINGTON - Every American should have health care coverage within
six years, Democratic Sen. Barack Obama said Thursday as he set an
ambitious goal soon after jumping into the 2008 presidential race.
"The time has come for universal health care in
America," Obama said at a conference of Families USA, a health care
advocacy group.
"I am absolutely determined that by the end of the first term of the
next president, we should have universal health care in this
country," the Illinois senator said.
Obama was previewing what is shaping up to be a
theme of the 2008 Democratic primary. His chief rivals, Sen. Hillary
Rodham Clinton and John Edwards, also are strong proponents of
universal health care and have promised to offer their plans.
Obama said while plans are offered in every
campaign season with "much fanfare and promise," they collapse under
the weight of Washington politics, leaving citizens to struggle with
the skyrocketing costs.
He said it's wrong that 46 million in this
country are uninsured when the country spends more than any one else
on health care. He said Americans pay $15 billion in taxes to help
care for the uninsured.
"We can't afford another disappointing charade in
2008, 2009 and 2010," Obama said. "It's not only tiresome, it's
wrong."
Obama's call was an echo of a speech he made last
April when he said Democrats "need to cling to the core values that
make us Democrats, the belief in universal health care, the belief
in universal education, and then we should be agnostic in terms of
how to achieve those values."
His argument Thursday not only will be considered
through the prism of the presidential campaign, but weighed against
rival Sen. Hillary Rodham Clinton's ill-fated plan to overhaul the
health care insurance system when she was first lady.
Even after leading that calamitous attempt in
1993, Clinton remains in favor of universal health care and has made
it a central theme of her presidential bid.
"One of the goals that I will be presenting ...
is health insurance for every child and universal health care for
every American," she said at a community health clinic in New York
Sunday, the day after entering the 2008 Democratic field. "That's a
very major part of my campaign and I want to hear people's ideas
about how we can achieve that goal."
She criticized Bush's proposal to make health
care more affordable through tax breaks, arguing that it would lead
to less funding for hospitals.
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Clinton was self-deprecating in describing her own experience in the
health care debate and joked that Bush would need some heavy-duty
protection as he wades into the fight.
"I welcome his participation in the health care
debate. I'm going to send him a suit of armor because I know anybody
who puts a foot in the health care debate is gonna need that. I've
got the scars and experience to show for it," said the New York
senator.
Another candidate, Ohio Rep. Dennis Kucinich,
also backs universal health care.
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Blacks at Higher Risk for Death
from Heart Failure
NHLBI Study
The results of a new study suggest that black patients with
congestive heart failure are at higher risk for death and for
worsening of their disease than similarly treated white patients.
"Congestive heart failure is a devastating disease affecting
600,000 black patients. If research confirms a difference between
black and white survival and identifies the underlying cause of this
difference, we may see improved treatment of blacks with heart
failure and a greater understanding of this disease among all
patients," said Dr. Claude Lenfant, Director of the National Heart,
Lung, and Blood Institute (NHBLI), which funded the study.
The new findings, published in the February 25 issue of the "New
England Journal of Medicine", are based on data collected from the
Studies of Left Ventricular Dysfunction (SOLVD). SOLVD found that
patients with chronic congestive heart failure had fewer deaths and
hospitalizations when treated with an ACE inhibitor, a drug which
blocks the constriction of blood vessels.
In the new "retrospective" analysis, Dr. Daniel Dries of the
NHLBI and colleagues looked back at the SOLVD results and analyzed
racial differences. They reported that 42 percent of blacks in the
treatment component of the study (patients with symptoms) and 22
percent of blacks in the prevention component (without symptoms)
died compared to 36 and 14 percent of whites respectively. In
addition, black patients were at increased risk for progression of
the disease.
Although other research has shown that the death rate for
congestive heart failure is more than twice as high in black
patients as in whites, not all studies have come to this conclusion.
Some studies have attributed differences in death rates to factors
such as differences in access to care or differences in the severity
of the disease. Other studies have not found a racial difference.
In the new study, the higher risk of death and disease
progression among black patients was found even after the scientists
adjusted the data to minimize any influences from age, other
coexisting medical conditions, severity and causes of heart failure,
socioeconomic status, and medications. Also, since SOLVD is a
clinical trial, management and follow-up of patients was
standardized.
Dr. Dries and his colleagues speculate that the differences
between blacks and whites with heart failure may be due to
physiological differences in the neuroendocrine and renin/angiotensinogen
systems. These systems release various hormones affecting heart rate
and blood flow in response to the heart's decreased ability to pump
blood. Over time, it is believed that the initial beneficial effect
of these hormones is stopped and instead they actually contribute to
the progression of heart failure.
The neuroendocrine system governs "catecholamines," a group of
hormones that includes norepinephrine; the renin/angiotensinogen
system involves the release of angiotensin II.
Black patients with heart failure may have a greater activation
of the neuroendocrine rather than the renin/angiotensinogen system,
suggest the study's authors. Another possible explanation is that
ACE inhibitors, which block the renin/angiotensinogen system, may be
less effective in halting the progression of the disease in black
compared to white patients. ACE inhibitors are commonly used to
treat heart failure.
If future studies confirm this racial difference, there may be
implications for improved treatment of black patients with heart
failure.
For example, some previous studies have suggested that when used
alone for the treatment of hypertension, ACE inhibitors may be less
effective in black patients. Beta-blockers, on the other hand,
antagonize or "block" the neuroendocrine system. If further research
finds that the neuroendocrine system is overactive in black
patients, they may be ideal candidates for early therapy combining
beta blockers with ACE inhibitors.
"The stakes are high -- over 4 1/2 million people have congestive
heart failure and hospitalizations for this disease are on the
rise," said Dr. Lenfant. "The NHLBI is committed to conducting
further research not only to confirm this apparent racial difference
but also to find ways to improve the treatment of all patients with
heart failure."
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Obesity
Tied to Worsening Kidney Function in Kids
RENO, NEV. — Increased body mass index is
correlated with worsening proteinuria in children, according
to study findings presented at the annual meeting of the
American College of Nutrition.
The data confirm the hypothesis that obesity contributes
to a decline in the glomerular filtration rate, an indicator
of kidney function, said Dr. Carolyn Abitbol of the Division
of Pediatric Nephrology at the University of Miami.
The study included 40 obese children: 16 low-birth-weight
children (less than 1,200 grams) and 24 children of normal
birth weight (more than 2,500 grams). Obesity was defined as
a body mass index (BMI) of greater than the 95th percentile
for age and gender. The study also included 20 nonobese
children of normal birth weight as clinical controls. All of
the children in the study had proteinuric kidney disease.
The researchers excluded any patients who had acute
glomerulonephritis immune-mediated nephritis, overt
diabetes, as well as patients who had HIV nephropathy.
Dr. Abitbol and colleagues performed kidney biopsies of
nine children in the obese, low-birth-weight group; all had
focal segmental glomerulosclerosis (FSGS).
Of the children in the normal birth weight, obese group,
16 were biopsied and 14 had FSGS. One patient had focal
mesangial proliferative glomerulonephropathy, probably an
early form of FSGS. Another had a membranous nephropathy,
which is unique and immune mediated. In the nonobese control
group, all of the children were biopsied and 14 had FSGS, 4
had mesangial glomerulonephropathy, 1 had minimal change,
and 1 was membranous.
Researchers compared the mean renal survival relative
with birth weight and found low-birth-weight patients had a
loss of glomerular filtration rate significantly earlier
than those of normal birth weight.
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